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1. They have a fixed alcohol receptor gene allowing their livers to once again function at a normal rate (alcohol affects the rate of liver function, increasing it slightly and causing the liver to age slightly faster while alcohol is above a certain level in their system). The defective version of this gene in other CFFs completely crippled their ability to properly turn nutrition into long-term stores or replenish these stores when they got depleted (unless they were drunk all the time), which would make them more dependent on eating regularly, and cause the 'near death' music to play when glycogen stores get low, and then to keep playing no matter how well they ate, because these stores would never get replenished. 2. Glucose creation from starch in foods lowered to normal value. This should make it a bit harder for them to gain weight too quickly. 3. Heart adipose receptor unchanged for now. These can still get heart disease... Hopefully the above changes make it so they can't easily get this fat. This needs testing... watch their adipose levels, and watch their heart health if these get really high (95% or more). 4. Pigment & pigment bleed gene fixes... cutting AND duplication now disabled for these genes to avoid duplicated pigment genes contributing to unnecessary extra problems with high-gens, as discussed in the CFF thread. |
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Yeah with the heart disease gene silenced too, all their fixes (as of CFF 1.1) should be working perfectly, other than obviously them not being able to get heart disease... The new ones will probably have some fixed more realistic genetics involving how they deal with hunger and use and store nutrition. Also possibly some differences in mating behavior that make them more picky when choosing mates. Whether or not the new ones will be able to get heart disease is still up in the air, depending on testing with the hunger/nutrition changes and how feasible it is to balance the genes. | |||||||||||
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Them some touchy genetics. At least it has a temporary fix. I was throwing together a breed based on these guys and they're doing great with the gene silenced. | |||||||||||
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Yeah, these guys won't survive really with that receptor... turns out the problem with the liver gene I fixed in them was masking the problem with the heart gene and normal adipose levels... | |||||||||||
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In my testing the adipose tissue level got really high and they rarely made it past half an hour. Edit: I turned off the receptor that damaged the heart and they're living just fine. | |||||||||||
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starch -> glucose -> pyruvate -> fatty acid -> triglyceride -> adipose... Pyruvate acts as a short term energy store, some of whatever is not needed gets turned back into glucose and then to glycogen, but much of this excess gets turned into fatty acids which make their way to adipose tissue. Of course starch is not the only way glucose gets into them, since it can come from metabolizing fatty acids from fat as well (fat->fatty acids->pyruvate->glucose), and all these things cycle back and forth according to need, so extra glucose not needed for making into pyruvate turns into glycogen as well. The chemical pathways in Creatures digestive systems are very well-modeled after real ones... they can definitely get fat from getting a lot of carbs, just like us... once glycogen gets to a certain threshold, pretty much all extra unneeded glucose getting into their systems from any kind of food after that will turn to fat. There was a neat chart showing all the workings of their metabolisms somewhere, one of the manuals I think, but it could have been the CDN site. | |||||||||||
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I don't think there is a pathway for starch to lead to adipose tissue, but I will make a thorough check. | |||||||||||
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Welp, these did fix the liver problems that stopped them from replenishing glycogen which caused the music issue, but now they tend to get obese and die of heart failure. I was worried about that. | |||||||||||
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